Chaojie Li

Neurosurgery Master's graduate from Southwest Medical University with a passion for scientific research, specifically in stroke science. Actively involved in cerebrovascular disease and neurodegeneration research, resulting in multiple publications and significant contributions to the field. Seeking research opportunities to expand expertise and make impactful strides in stroke research.

• World Stroke Organization (ESO-WSO-Conference)
• Meeting of Neurosurgical Cerebrovascular Diseases Group of Sichuan Medical Association
• World Federation of Neurosurgical Societies Symposium (WFNS)
• The Third Academic Conference on Neuropathy in Sichuan Province
• Forum for Academic Exchange of Young Scholars of Southwest Medical University

• Qianke Tao, Xiancheng Qiu, Chaojie Li, et al., S100A8 regulates autophagy-dependent ferroptosis in microglia after experimental subarachnoid hemorrhage (Experimental Neurology)
• Chaojie Li, Peng Lu, Lihan Zhang, et al., Apolipoprotein E polymorphism impacts white matter injury through microglial phagocytosis after experimental subarachnoid hemorrhage (Neuroscience)
• Lihan Zhang, Chaojie Li (co-author), Yijing He, et al., TRPM4 drives cerebral edema by switching to alternative splicing isoform after experimental traumatic brain injury (Journal of Neurotrauma)
• Jianhua Peng, Yijing He, Kecheng Guo, Jinwei Pang, Ghosh Dipritu, Lihan Zhang, Long Gu, Yuke Xie, Zheng Bao, Qianke Tao, Xiancheng Qiu, Tianqi Tu, Chenghao Kuang, Jian Zhou, Chaojie Li, Xi Kong, Lifang Zhang, Shigang Yin, Yong Jiang, Multi-omics reveals molecular linkages associated with neurodegeneration after acute ischemic stroke (eBioMedicine)

An integrated multi-omics study to identify dynamic molecular alterations associated with acute brain injury

• Autophagy-dependent ferroptosis in microglia after experimental subarachnoid hemorrhage
• Alternative splicing isoform in experimental traumatic brain injury
• Multi-omics reveals molecular linkages associated with neurodegeneration after acute ischemic stroke
• Microglial phagocytosis impacts white matter injury after experimental subarachnoid hemorrhage
• Mechanism of mitochondrial homeostasis affected by histone H3K23 lactic acid modification after SAH
• Epigenetic regulation of SARS-Cov-2 spike protein modulates cellular viability and nascent RNA transcription in neuronal cells
• Improving neuronal injury by inhibiting mitochondrial fragmentation of astrocytes after experimental subarachnoid hemorrhage
• Modulate nerve injury by regulating the quality of astrocyte Mitochondrial in hemorrhagic stroke
• Construction and accurate intervention of cerebrovascular disease brain model based on human pluripotent Stem cells