Vineet Arora

• 2nd Place – Oral Presentation, Scripps Research Fest (2021)
• Leon and Friends Foundation Postdoctoral Fellowship (2019)
• Travel Award, European College of Neuropsychopharmacology (ECNP) (2018)
• FENS–ELECTRAIN Training Award, Federation of European Neuroscience Societies (FENS) (2018)
• Severo Ochoa Predoctoral Fellowship, Spanish Research Council (2015)
• Graduate School Scholarship, International Max Planck Research School, Tübingen, Germany (2012)

• Arora, V., Rossano, A. J., Frederick, D. J., Anderson, S., et al. (in prep) Two fuels, one synapse: demand-dependent metabolic specialization governs vesicle recycling and transmission fidelity in human stem cell-derived neurons.
• Rossano, A. J., Arora, V., Frederick, D. J., Kim, M., Sankuratri, Y., Anderson, S., et al. (in prep) Impaired synaptic vesicle docking kinetics in 22q11.2 deletion syndrome human iPSC-derived neurons revealed by iGluSnFR and pHluorin imaging.
• Arora, V., Bernhardt, A., Napoli, A., & Serruya, M. (2025). Enhancing neuronal viability: The protective role of 10% human cerebrospinal fluid in primary neuronal cultures. Brain Research, 1864, 149782. https://doi.org/10.1016/j.brainres.2025.149782 Demonstrated neuroprotective effects of human CSF in primary neuronal cultures, revealing enhanced neuronal survival under optimized supplementation conditions.
• Kilinc, M., Arora, V., Creson, T. K., et al. (2022). Endogenous Syngap1 alpha splice forms promote cognitive function and seizure protection. eLife, 11, e75707. https://doi.org/10.7554/eLife.75707 Identified SYNGAP1 splice isoforms as key regulators of cognitive function and seizure resilience through molecular and functional neuroscience approaches.
• Llamosas, N., Arora, V., Vij, R., et al. (2020). SYNGAP1 controls the maturation of dendrites, synaptic function, and network activity in developing human neurons. Journal of Neuroscience, 40(41), 7980–7994. https://doi.org/10.1523/JNEUROSCI.1367-20.2020 Defined the role of SYNGAP1 in regulating dendritic development, synaptic maturation, and emergent network activity in human neuronal systems.
• Arora, V., Aller, M. I., & Lerma, J. (2019). Grik4 over-expression underlies altered synaptic gain and enhances basal activation of amygdala circuits. European Neuropsychopharmacology, 29.
• Lerma, J., Arora, V., Valbuena, S., García, A., & Aller, M. I. Kainate receptors, circuit imbalance and mental diseases. IBRO Reports, 6, S29.
• Arora, V., Pecoraro, V., Aller, M. I., et al. (2018). Increased Grik4 gene dosage causes imbalanced circuit output and human disease-related behaviors. Cell Reports, 23(13), 3827–3838. https://doi.org/10.1016/j.celrep.2018.05.086 Established that elevated GRIK4 dosage disrupts excitatory circuit balance, leading to disease-relevant behavioral phenotypes.
• Engelholm, M., Sekler, J., Schöndorf, D. C., Arora, V., et al. (2014). A novel mutation in LRSAM1 causes axonal Charcot-Marie-Tooth disease with dominant inheritance. BMC Neurology, 14, 118. https://doi.org/10.1186/1471-2377-14-118 Identified a pathogenic LRSAM1 mutation linked to axonal Charcot-Marie-Tooth disease with dominant inheritance.

Editorial & Peer Review Contributions

Editorial Roles

• Current Review Editor, Frontiers in Medicine

Peer Review Activities
Reviewer for peer-reviewed manuscripts in the following journals:

• Frontiers in Medicine
• Frontiers in Neuroscience
• Translational Medicine
• Scientific Reports
• Molecular and Cellular Neuroscience
• Neuroscience Letters
• IBRO Neuroscience Reports
• PLOS ONE
• Cellular Oncology